Ashmeetha Manilall
University of the Witwatersrand
South Africa
About
No AI summary yet.
Metrics
- Publications
- 19
- Citations
- 166
- h-index
- 6
- Publishing journals
- 18
- Years of publications
- 10
- Active since
- 2017
- References
- 976
- Institutions
- 1
Publication history
Latest publications
- Could exercise intercept the profibrotic and stiffening trajectory of HFpEF progression in a clinically faithful porcine LV pressure-overload model?
- Cytokine-mediated inflammatory responses in neonatal Group B Streptococcus disease and their role in neurodevelopmental impairment
- Involvement of microRNA-146a-5p, but not -155-5p and -29b-5p, in left ventricular remodeling and dysfunction in spontaneously hypertensive rats
- Acute exposure to LPS induces cardiac dysfunction via the activation of the NLRP3 inflammasome
- Involvement of pentraxin‐3 in the development of hypertension but not left ventricular hypertrophy in male spontaneously hypertensive rats
- The Cytotoxic Effects of Nyaope, a Heroin-based Street Drug, in SH-SY5Y Neuroblastoma Cells
- Tocilizumab does not ameliorate inflammation-induced left ventricular dysfunction in a collagen-induced arthritis rat model
- Alterations In Methylation And Snps Of Oprm1 Gene Show Lack Of Association With Heroin (Nyaope) Consumption In South African Males Of African Ancestry
- Interleukin-6 Blockers Improve Inflammation-Induced Lipid Metabolism Impairments but Induce Liver Fibrosis in Collagen-Induced Arthritis
- Tumor Necrosis Factor-α Mediates Inflammation-induced Early-Stage Left Ventricular Systolic Dysfunction
- TNF-α inhibitors reduce inflammation-induced concentric remodelling, but not diastolic dysfunction in collagen-induced arthritis.
- The effect of TNF-α inhibitor treatment on microRNAs and endothelial function in collagen induced arthritis
- Increased protein phosphatase 5 expression in inflammation-induced left ventricular dysfunction in rats
- Inflammation‐induced left ventricular fibrosis is partially mediated by tumor necrosis factor‐α
- Genetics of ncHH: from a peculiar inheritance of a novel GNRHR mutation to a comprehensive review of the literature
- Similarities and differences in the reproductive phenotypes of women with congenital hypogonadotrophic hypogonadism caused by GNRHR mutations and women with polycystic ovary syndrome
- Glu2.53(90) of the GnRH receptor is part of the conserved G protein-coupled receptor structure and does not form a salt-bridge with Lys3.32(121)
- Gonadotropin-releasing hormone (GnRH) receptor structure and GnRH binding
- The role of the GLU2.53(90) residue in gonadotropin-releasing hormone receptor expression and function
No publications in the index yet.